MDR-1, Bcl-xL, H. pylori, and Wnt/β-catenin signalling in the adult stomach: how much is too much?
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Molecular Biology,Pathology and Forensic Medicine
Link
http://www.nature.com/articles/labinvest2012151.pdf
Reference28 articles.
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2. Rocco A, Compare D, Liguroi E et al. MDR1-P-lgycoprotein behaves as an oncofetal protein that promotes cell survival in gastric cancer cells. Lab Invest, (this issue).
3. Asciutti S, Akiri G, Grumolato L et al. Diverse mechanisms of Wnt activation and effects of pathway inhibition on proliferation of human gastric carcinoma cells. Oncogene 2011;30:956–966.
4. Yamada T, Takaoka AS, Naishiro Y et al. Transactivation of the multidrug resistance 1 gene by T-cell factor 4/beta-catenin complex in early colorectal carcinogenesis. Cancer Res 2000;60:4761–4766.
5. Clevers H, Nusse R . Wnt/β-catenin signaling and disease. Cell 2012;149:1192–1205.
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1. H. pylori slyD, a novel virulence factor, is associated with Wnt pathway protein expression during gastric disease progression;Microbial Pathogenesis;2020-11
2. Galangin (GG) combined with cisplatin (DDP) to suppress human lung cancer by inhibition of STAT3-regulated NF-κB and Bcl-2/Bax signaling pathways;Biomedicine & Pharmacotherapy;2018-01
3. An increase in glucosylceramide synthase induces Bcl-xL-mediated cell survival in vinorelbine-resistant lung adenocarcinoma cells;Oncotarget;2015-05-12
4. Expression of the E-cadherin/β-catenin/tcf-4 Pathway in Gastric Diseases with Relation to Helicobacter pylori Infection: Clinical and Pathological Implications;Asian Pacific Journal of Cancer Prevention;2014-01-15
5. α-Lipoic Acid InhibitsHelicobacter pylori-Induced Oncogene Expression and Hyperproliferation by Suppressing the Activation of NADPH Oxidase in Gastric Epithelial Cells;Mediators of Inflammation;2014
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