GCN2 kinase is a key regulator of fibrogenesis and acute and chronic liver injury induced by carbon tetrachloride in mice-Reference-Cited by-同舟云学术

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GCN2 kinase is a key regulator of fibrogenesis and acute and chronic liver injury induced by carbon tetrachloride in mice

Published:2013-01-14 Issue:3 Volume:93 Page:303-310
ISSN:0023-6837
Container-title:Laboratory Investigation
language:en
Short-container-title:Lab Invest

Author:

Arriazu Elena,Ruiz de Galarreta Marina,López-Zabalza María J,Leung Tung Ming,Nieto Natalia,Iraburu María J

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology,Pathology and Forensic Medicine

Link

http://www.nature.com/articles/labinvest2012173.pdf

Reference33 articles.

1. Wek SA, Zhu S, Wek RC . The histidyl-tRNA synthetase-related sequence in the eIF-2 alpha protein kinase GCN2 interacts with tRNA and is required for activation in response to starvation for different amino acids. Mol Cell Biol 1995;15:4497–4506.

2. Dong J, Qiu H, Garcia-Barrio M et al. Uncharged tRNA activates GCN2 by displacing the protein kinase moiety from a bipartite tRNA-binding domain. Mol Cell 2000;6:269–279.

3. Kilberg MS, Shan J, Su N . ATF4-dependent transcription mediates signaling of amino acid limitation. Trends Endocrinol Metab 2009;20:436–443.

4. Kimball SR, Jefferson LS . Role of amino acids in the translational control of protein synthesis in mammals. Semin Cell Dev Biol 2005;16:21–27.

5. Berlanga JJ, Ventoso I, Harding HP et al. Antiviral effect of the mammalian translation initiation factor 2alpha kinase GCN2 against RNA viruses. EMBO J 2006;25:1730–1740.

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