Nuclear factor-κB2/p100 promotes endometrial carcinoma cell survival under hypoxia in a HIF-1α independent manner-Reference-Cited by-同舟云学术

Nuclear factor-κB2/p100 promotes endometrial carcinoma cell survival under hypoxia in a HIF-1α independent manner

Published:2011-05-02 Issue:6 Volume:91 Page:859-871
ISSN:0023-6837
Container-title:Laboratory Investigation
language:en
Short-container-title:Lab Invest

Author:

Yeramian Andree,Santacana Maria,Sorolla Anabel,Llobet David,Encinas Mario,Velasco Ana,Bahi Nuria,Eritja Nuria,Domingo Mónica,Oliva Esther,Dolcet Xavier,Matias-Guiu Xavier

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology,Pathology and Forensic Medicine

Link

http://www.nature.com/articles/labinvest201158.pdf

Reference44 articles.

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3. Vaupel P, Kelleher DK, Hockel M . Oxygen status of malignant tumors: pathogenesis of hypoxia and significance for tumor therapy. Semin Oncol 2001;28 (2 Suppl 8):29–35.

4. Rius J, Guma M, Schachtrup C, et al. NF-kappaB links innate immunity to the hypoxic response through transcriptional regulation of HIF-1alpha. Nature 2008;453:807–811.

5. Kazi AA, Koos RD . Estrogen-induced activation of hypoxia-inducible factor-1alpha, vascular endothelial growth factor expression, and edema in the uterus are mediated by the phosphatidylinositol 3-kinase/Akt pathway. Endocrinology 2007;148:2363–2374.

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