Transient ischemia/hypoxia enhances gentamicin ototoxicity via caspase-dependent cell death pathway-Reference-Cited by-同舟云学术

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Transient ischemia/hypoxia enhances gentamicin ototoxicity via caspase-dependent cell death pathway

Published:2011-04-25 Issue:7 Volume:91 Page:1092-1106
ISSN:0023-6837
Container-title:Laboratory Investigation
language:en
Short-container-title:Lab Invest

Author:

Lin Chia-Der,Kao Ming-Ching,Tsai Ming-Hsui,Lai Chih-Ho,Wei I-Hua,Tsai Mang-Hung,Tang Chih-Hsin,Lin Cheng-Wen,Hsu Chuan-Jen,Lin Ching-Yuang

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology,Pathology and Forensic Medicine

Link

http://www.nature.com/articles/labinvest201169.pdf

Reference60 articles.

1. Gupta R, Kim JY, Espinal MA, et al. Public health. Responding to market failures in tuberculosis control. Science 2001;293:1049–1051.

2. Forge A, Schacht J . Aminoglycoside antibiotics. Audiol Neurootol 2000;5:3–22.

3. Contopoulos-Ioannidis DG, Giotis ND, Baliatsa DV, et al. Extended-interval aminoglycoside administration for children: a meta-analysis. Pediatrics 2004;114:e111–e118.

4. Bernard PA . Freedom from ototoxicity in aminoglycoside treated neonates: a mistaken notion. Laryngoscope 1981;91:1985–1994.

5. Ensink RJ, Camp GV, Cremers CW . Mitochondrial inherited hearing loss. Clin Otolaryngol Allied Sci 1998;23:3–8.

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