Diabetic eNOS knockout mice develop distinct macro- and microvascular complications-Reference-Cited by-同舟云学术

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Diabetic eNOS knockout mice develop distinct macro- and microvascular complications

Published:2008-04-07 Issue:5 Volume:88 Page:515-528
ISSN:0023-6837
Container-title:Laboratory Investigation
language:en
Short-container-title:Lab Invest

Author:

Mohan Sumathy,Reddick Robert L,Musi Nicolas,Horn Diane A,Yan Bo,Prihoda Thomas J,Natarajan Mohan,Abboud-Werner Sherry L

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology,Pathology and Forensic Medicine

Link

http://www.nature.com/articles/labinvest200823.pdf

Reference34 articles.

1. Hink U, Li H, Mollnau H, et al. Mechanisms underlying endothelial dysfunction in diabetes mellitus. Circ Res 2001;88:E14–E22.

2. Hoshiyana M, Li B, Yao J . Effect of high glucose on nitric oxide production and endothelial nitric oxide synthase protein expression in human glomerular endothelial cells. Nephron Exp Nephrol 2003;95:e62–e68.

3. Huang PL . Endothelial nitric oxide synthase and endothelial dysfunction. Curr Hypertens Rep 2003;5:473–480.

4. Deng A, Baylis C . Locally produced EDRF controls preglomerular resistance and ultrafiltration coefficient. Am J Physiol Renal Fluid Electrolyte Physiol 1993;264:F212–F215.

5. Zatz R, de Nucci G . Effects of acute nitric oxide inhibition on rat glomerular microcirculation. Am J Physiol Renal Fluid Electrolyte Physiol 1991;261:F360–F363.

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4. Deletion, but Not Heterozygosity, of eNOS Raises Blood Pressure and Aggravates Nephropathy in BTBR ob/ob Mice;Nephron;2024-02-01

5. Angiotensin II type 1 receptor-associated protein deletion combined with angiotensin II stimulation accelerates the development of diabetic kidney disease in mice on a C57BL/6 strain;Hypertension Research;2023-11-13

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