A dominant autoinflammatory disease caused by non-cleavable variants of RIPK1-Reference-Cited by-同舟云学术

A dominant autoinflammatory disease caused by non-cleavable variants of RIPK1

Published:2019-12-11 Issue:7788 Volume:577 Page:109-114
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Tao Panfeng,Sun Jinqiao,Wu Zheming,Wang Shihao,Wang Jun,Li Wanjin,Pan Heling,Bai Renkui,Zhang Jiahui,Wang Ying,Lee Pui Y.,Ying Wenjing,Zhou Qinhua,Hou Jia,Wang Wenjie,Sun Bijun,Yang Mi,Liu Danru,Fang Ran,Han Huan,Yang Zhaohui,Huang Xin,Li Haibo,Deuitch Natalie,Zhang Yuan,Dissanayake Dilan,Haude Katrina,McWalter Kirsty,Roadhouse Chelsea,MacKenzie Jennifer J.,Laxer Ronald M.,Aksentijevich Ivona,Yu Xiaomin,Wang Xiaochuan,Yuan Junying,Zhou Qing

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41586-019-1830-y.pdf

Reference28 articles.

1. Yuan, J., Amin, P. & Ofengeim, D. Necroptosis and RIPK1-mediated neuroinflammation in CNS diseases. Nat. Rev. Neurosci. 20, 19–33 (2019).

2. Zhang, X., Dowling, J. P. & Zhang, J. RIPK1 can mediate apoptosis in addition to necroptosis during embryonic development. Cell Death Dis. 10, 245 (2019).

3. Newton, K. et al. Cleavage of RIPK1 by caspase-8 is crucial for limiting apoptosis and necroptosis. Nature 574, 428–431 (2019).

4. Xu, D. et al. TBK1 suppresses RIPK1-driven apoptosis and inflammation during development and in aging. Cell 174, 1477–1491 (2018).

5. Meng, H. et al. Death-domain dimerization-mediated activation of RIPK1 controls necroptosis and RIPK1-dependent apoptosis. Proc. Natl Acad. Sci. USA 115, E2001–E2009 (2018).

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