Integration of innate immune signalling by caspase-8 cleavage of N4BP1-Reference-Cited by-同舟云学术

Integration of innate immune signalling by caspase-8 cleavage of N4BP1

Published:2020-09-24 Issue:7833 Volume:587 Page:275-280
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Gitlin Alexander D.^ORCID,Heger Klaus,Schubert Alexander F.,Reja Rohit,Yan Donghong,Pham Victoria C.,Suto Eric,Zhang Juan,Kwon Youngsu C.,Freund Emily C.,Kang Jing,Pham Anna,Caothien Roger,Bacarro Natasha,Hinkle Trent,Xu Min,McKenzie Brent S.,Haley Benjamin,Lee Wyne P.,Lill Jennie R.,Roose-Girma Merone,Dohse Monika,Webster Joshua D.,Newton Kim^ORCID,Dixit Vishva M.^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

https://www.nature.com/articles/s41586-020-2796-5.pdf

Reference53 articles.

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2. Fisher, G. H. et al. Dominant interfering Fas gene mutations impair apoptosis in a human autoimmune lymphoproliferative syndrome. Cell 81, 935–946 (1995).

3. Del-Rey, M. et al. A homozygous Fas ligand gene mutation in a patient causes a new type of autoimmune lymphoproliferative syndrome. Blood 108, 1306–1312 (2006).

4. Chun, H. J. et al. Pleiotropic defects in lymphocyte activation caused by caspase-8 mutations lead to human immunodeficiency. Nature 419, 395–399 (2002).

5. Lehle, A. S. et al. Intestinal inflammation and dysregulated immunity in patients with inherited caspase-8 deficiency. Gastroenterology 156, 275–278 (2019).

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