OTULIN limits cell death and inflammation by deubiquitinating LUBAC-Reference-Cited by-同舟云学术

OTULIN limits cell death and inflammation by deubiquitinating LUBAC

Published:2018-06-27 Issue:7712 Volume:559 Page:120-124
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Heger Klaus,Wickliffe Katherine E.,Ndoja Ada,Zhang Juan,Murthy Aditya,Dugger Debra L.,Maltzman Allie,de Sousa e Melo Felipe,Hung Jeffrey,Zeng Yi,Verschueren Erik,Kirkpatrick Donald S.,Vucic Domagoj,Lee Wyne P.,Roose-Girma Merone,Newman Robert J.,Warming Søren,Hsiao Yi-Chun,Kőműves László G.,Webster Joshua D.,Newton Kim,Dixit Vishva M.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41586-018-0256-2.pdf

Reference32 articles.

1. Damgaard, R. B. et al. The deubiquitinase OTULIN is an essential negative regulator of inflammation and autoimmunity. Cell 166, 1215–1230.e20 (2016).

2. Zhou, Q. et al. Biallelic hypomorphic mutations in a linear deubiquitinase define otulipenia, an early-onset autoinflammatory disease. Proc. Natl Acad. Sci. USA 113, 10127–10132 (2016).

3. Rivkin, E. et al. The linear ubiquitin-specific deubiquitinase gumby regulates angiogenesis. Nature 498, 318–324 (2013).

4. Peltzer, N. et al. HOIP deficiency causes embryonic lethality by aberrant TNFR1-mediated endothelial cell death. Cell Reports 9, 153–165 (2014).

5. Keusekotten, K. et al. OTULIN antagonizes LUBAC signaling by specifically hydrolyzing Met1-linked polyubiquitin. Cell 153, 1312–1326 (2013).

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