Activity of caspase-8 determines plasticity between cell death pathways-Reference-Cited by-同舟云学术

Activity of caspase-8 determines plasticity between cell death pathways

Published:2019-11-13 Issue:7784 Volume:575 Page:679-682
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Newton Kim,Wickliffe Katherine E.,Maltzman Allie,Dugger Debra L.,Reja Rohit,Zhang Yue,Roose-Girma Merone,Modrusan Zora,Sagolla Meredith S.,Webster Joshua D.,Dixit Vishva M.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41586-019-1752-8.pdf

Reference38 articles.

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3. Kaiser, W. J. et al. RIP3 mediates the embryonic lethality of caspase-8-deficient mice. Nature 471, 368–372 (2011).

4. Alvarez-Diaz, S. et al. The pseudokinase MLKL and the kinase RIPK3 have distinct roles in autoimmune disease caused by loss of death-receptor-induced apoptosis. Immunity 45, 513–526 (2016).

5. Newton, K. et al. Cleavage of RIPK1 by caspase-8 is crucial for limiting apoptosis and necroptosis. Nature 574, 428–431 (2019).

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