Nuclear heparanase-1 activity suppresses melanoma progression via its DNA-binding affinity
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Genetics,Molecular Biology
Link
http://www.nature.com/articles/onc201540.pdf
Reference58 articles.
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3. Zong F, Fthenou E, Wolmer N, Hollosi P, Kovalszky I, Szilak L et al. Syndecan-1 and FGF-2, but not FGF receptor-1, share a common transport route and co-localize with heparanase in the nuclei of mesenchymal tumor cells. PLoS ONE 2009; 4: e7346.
4. Purushothaman A, Hurst DR, Pisano C, Mizumoto S, Sugahara K, Sanderson RD . Heparanase-mediated loss of nuclear syndecan-1 enhances histone acetyltransferase (HAT) activity to promote expression of genes that drive an aggressive tumor phenotype. J Biol Chem 2011; 286: 30377–30383.
5. Purushothaman A, Chen L, Yang Y, Sanderson RD . Heparanase stimulation of protease expression implicates it as a master regulator of the aggressive tumor phenotype in myeloma. J Biol Chem 2008; 283: 32628–32636.
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