Melanocyte transformation requires complete loss of all pocket protein function via a mechanism that mitigates the need for MAPK pathway activation-Reference-Cited by-同舟云学术

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Melanocyte transformation requires complete loss of all pocket protein function via a mechanism that mitigates the need for MAPK pathway activation

Published:2017-02-13 Issue:26 Volume:36 Page:3789-3795
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Tonks I D,Mukhopadhyay P,Schroder W A,Sorolla A,Mould A W,Handoko H Y,Ferguson B,Muller H K,Keith P,Hayward N K,Walker G J,Kay G F

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

https://www.nature.com/articles/onc2016511.pdf

Reference36 articles.

1. Hanahan D, Weinberg RA . Hallmarks of cancer: the next generation. Cell 2011; 144: 646–674.

2. Zarkowska T, Mittnacht S . Differential phosphorylation of the retinoblastoma protein by G1/S cyclin-dependent kinases. J Biol Chem 1997; 272: 12738–12746.

3. Hansen K, Farkas T, Lukas J, Holm K, Rönnstrand L, Bartek J . Phosphorylation-dependent and -independent functions of p130 cooperate to evoke a sustained G1 block. EMBO J 2001; 20: 422–432.

4. Leng X, Noble M, Adams PD, Qin J, Harper JW . Reversal of growth suppression by p107 via direct phosphorylation by cyclin D1/cyclin-dependent kinase 4. Mol Cell Biol 2002; 22: 2242–2254.

5. Grana X, Garriga J, Mayol X . Role of the retinoblastoma protein family, pRB, p107 and p130 in the negative control of cell growth. Oncogene 1998; 17: 3365–3383.

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