Fen1 mutations that specifically disrupt its interaction with PCNA cause aneuploidy-associated cancer-Reference-Cited by-同舟云学术

Fen1 mutations that specifically disrupt its interaction with PCNA cause aneuploidy-associated cancer

Published:2011-03-08 Issue:7 Volume:21 Page:1052-1067
ISSN:1001-0602
Container-title:Cell Research
language:en
Short-container-title:Cell Res

Author:

Zheng Li,Dai Huifang,Hegde Muralidhar L,Zhou Mian,Guo Zhigang,Wu Xiwei,Wu Jun,Su Lei,Zhong Xueyan,Mitra Sankar,Huang Qin,Kernstine Kemp H,Pfeifer Gerd P,Shen Binghui

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Molecular Biology

Link

http://www.nature.com/articles/cr201135.pdf

Reference56 articles.

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3. Liu Y, Kao HI, Bambara RA . Flap endonuclease 1: a central component of DNA metabolism. Annu Rev Biochem 2004; 73:589–615.

4. Parrish JZ, Yang C, Shen B, Xue D . CRN-1, a Caenorhabditis elegans FEN-1 homologue, cooperates with CPS-6/EndoG to promote apoptotic DNA degradation. EMBO J 2003; 22:3451–3460.

5. Singh P, Zheng L, Chavez V, Qiu J, Shen B . Concerted action of exonuclease and Gap-dependent endonuclease activities of FEN-1 contributes to the resolution of triplet repeat sequences (CTG)n- and (GAA)n-derived secondary structures formed during maturation of Okazaki fragments. J Biol Chem 2007; 282:3465–3477.

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