White spot syndrome virus entry is dependent on multiple endocytic routes and strongly facilitated by Cq-GABARAP in a CME-dependent manner-Reference-Cited by-同舟云学术

White spot syndrome virus entry is dependent on multiple endocytic routes and strongly facilitated by Cq-GABARAP in a CME-dependent manner

Published:2016-07 Issue:1 Volume:6 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Chen Rong-yuan,Shen Kai-li,Chen Zhen,Fan Wei-wei,Xie Xiao-lu,Meng Chuang,Chang Xue-jiao,Zheng Li-bing,Jeswin Joseph,Li Cheng-hua,Wang Ke-jian,Liu Hai-peng

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep28694.pdf

Reference56 articles.

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2. Shekhar, M. S. & Ponniah, A. G. Recent insights into host-pathogen interaction in white spot syndrome virus infected penaeid shrimp. J Fish Dis 38, 599–612 (2015).

3. Huang, H. T., Leu, J. H., Huang, P. Y. & Chen, L. L. A putative cell surface receptor for white spot syndrome virus is a member of a transporter superfamily. PLoS One 7, e33216 (2012).

4. Huang, H. T., Chan, H. L., Shih, T. Y. & Chen, L. L. A study of the role of glucose transporter 1 (Glut1) in white spot syndrome virus (WSSV) infection. Fish Shellfish Immunol 46, 305–314 (2015).

5. Wang, X. W., Xu, Y. H., Xu, J. D., Zhao, X. F. & Wang, J. X. Collaboration between a soluble C-type lectin and calreticulin facilitates white spot syndrome virus infection in shrimp. J Immunol 193, 2106–2117 (2014).

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