Dissociation of muscle insulin sensitivity from exercise endurance in mice by HDAC3 depletion-Reference-Cited by-同舟云学术

Dissociation of muscle insulin sensitivity from exercise endurance in mice by HDAC3 depletion

Published:2016-12-19 Issue:2 Volume:23 Page:223-234
ISSN:1078-8956
Container-title:Nature Medicine
language:en
Short-container-title:Nat Med

Author:

Hong Sungguan,Zhou Wenjun,Fang Bin,Lu Wenyun,Loro Emanuele,Damle Manashree,Ding Guolian,Jager Jennifer,Zhang Sisi,Zhang Yuxiang,Feng Dan,Chu Qingwei,Dill Brian D,Molina Henrik,Khurana Tejvir S,Rabinowitz Joshua D,Lazar Mitchell A,Sun Zheng

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://www.nature.com/articles/nm.4245.pdf

Reference64 articles.

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2. Samuel, V.T. & Shulman, G.I. Mechanisms for insulin resistance: common threads and missing links. Cell 148, 852–871 (2012).

3. Dubé, J.J. et al. Exercise-induced alterations in intramyocellular lipids and insulin resistance: the athlete's paradox revisited. Am. J. Physiol. Endocrinol. Metab. 294, E882–E888 (2008).

4. Brüning, J.C. et al. A muscle-specific insulin receptor knockout exhibits features of the metabolic syndrome of NIDDM without altering glucose tolerance. Mol. Cell 2, 559–569 (1998).

5. Kim, J.K. et al. Redistribution of substrates to adipose tissue promotes obesity in mice with selective insulin resistance in muscle. J. Clin. Invest. 105, 1791–1797 (2000).

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