Transfection of rat or mouse neurons by biolistics or electroporation-Reference-Cited by-同舟云学术

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Transfection of rat or mouse neurons by biolistics or electroporation

Published:2009-07-09 Issue:8 Volume:4 Page:1118-1127
ISSN:1754-2189
Container-title:Nature Protocols
language:en
Short-container-title:Nat Protoc

Author:

Dib-Hajj Sulayman D,Choi Jin Sung,Macala Lawrence J,Tyrrell Lynda,Black Joel A,Cummins Theodore R,Waxman Stephen G

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology

Link

http://www.nature.com/articles/nprot.2009.90.pdf

Reference58 articles.

1. Choi, J., Hudmon, A., Waxman, S. & Dib-Hajj, S. Calmodulin regulates current density and frequency-dependent inhibition of sodium channel Nav1.8 in DRG neurons. J. Neurophysiol. 96, 97–108 (2006).

2. Cummins, T.R. et al. Nav1.3 sodium channels: rapid repriming and slow closed-state inactivation display quantitative differences after expression in a mammalian cell line and in spinal sensory neurons. J. Neurosci. 21, 5952–5961 (2001).

3. Cummins, T.R., Dib-Hajj, S.D., Herzog, R.I. & Waxman, S.G. Nav1.6 channels generate resurgent sodium currents in spinal sensory neurons. FEBS Lett. 579, 2166–2170 (2005).

4. Dib-Hajj, S.D. et al. Gain-of-function mutation in Nav1.7 in familial erythromelalgia induces bursting of sensory neurons. Brain 128, 1847–1854 (2005).

5. Dib-Hajj, S.D. et al. Paroxysmal extreme pain disorder M1627K mutation in human Nav1.7 renders DRG neurons hyperexcitable. Mol. Pain 4, 37 (2008).

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