Genetic polymorphisms of N-acetyltransferase 1 and 2 and risk of cigarette smoking-related bladder cancer

Author:

Hsieh F-I,Pu Y-S,Chern H-D,Hsu L-I,Chiou H-Y,Chen C-J

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Oncology

Reference32 articles.

1. Augustine, A, Hebert, JR, Kabat, GC & Wynder, EL (1988). Bladder cancer in relation to cigarette smoking. Cancer Res 48: 4405–4408.

2. Bartsch, H, Malaveille, C, Friesen, M, Kadlubar, FF & Vineis, P (1993). Black (ari-cured) and blond (flue-cured) tobacco cancer risk IV: molecular dosimetry studies implicate aromatic amines as bladder carcinogens. Eur J Cancer 29A: 1199–1207.

3. Bell, DA, Taylor, JA, Butler, MA, Stephens, E, Wiest, J, Brubaker, L, Kadlubar, FF & Lucier, GW (1993). Genotype/phenotype discordance for human arylamine N-acetyltransferase (NAT2) reveals a new slow-acetylator allele common in African-Americans. Carcinogenesis 14: 1689–1692.

4. Bell, DA, Badawi, AF, Lang, NP, Ilett, KF, Kadlubar, FF & Hirvonen, A (1995a). Polymorphism in the N-acetyltransferase 1 (NAT1) polyadenylation signal: association of NAT1*10 allele with higher N-acetylation activity in bladder and colon tissue. Cancer Res 55: 5226–5229.

5. Bell, DA, Stephens, EA, Castranio, T, Umbach, DM, Watson, M, Deakin, M, Elder, J & Berkowitz, RS (1995b). Polyadenylation polymorphism in the acetyltransferase 1 gene (NAT1) increases risk of colorectal cancer. Cancer Res 55: 3537–3542.

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