Metabolic reprogramming by PCK1 promotes TCA cataplerosis, oxidative stress and apoptosis in liver cancer cells and suppresses hepatocellular carcinoma-Reference-Cited by-同舟云学术

Metabolic reprogramming by PCK1 promotes TCA cataplerosis, oxidative stress and apoptosis in liver cancer cells and suppresses hepatocellular carcinoma

Published:2018-01-16 Issue:12 Volume:37 Page:1637-1653
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Liu Meng-Xi,Jin Lei,Sun Si-Jia,Liu Peng,Feng Xu,Cheng Zhou-Li,Liu Wei-Ren,Guan Kun-Liang^ORCID,Shi Ying-Hong,Yuan Hai-Xin,Xiong Yue

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/s41388-017-0070-6.pdf

Reference32 articles.

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3. Warburg O. On the origin of cancer cells. Science. 1956;123:309–14.

4. Burgess SC, He T, Yan Z, Lindner J, Sherry AD, Malloy CR, et al. Cytosolic phosphoenolpyruvate carboxykinase does not solely control the rate of hepatic gluconeogenesis in the intact mouse liver. Cell Metab. 2007;5:313–20.

5. Yang J, Kalhan SC, Hanson RW. What is the metabolic role of phosphoenolpyruvate carboxykinase? J Biol Chem. 2009;284:27025–9.

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