Vav proteins maintain epithelial traits in breast cancer cells using miR-200c-dependent and independent mechanisms-Reference-Cited by-同舟云学术

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Vav proteins maintain epithelial traits in breast cancer cells using miR-200c-dependent and independent mechanisms

Published:2018-08-07 Issue:2 Volume:38 Page:209-227
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Lorenzo-Martín L. Francisco,Citterio Carmen,Menacho-Márquez Mauricio,Conde Javier,Larive Romain M.,Rodríguez-Fdez Sonia,García-Escudero Ramón^ORCID,Robles-Valero Javier,Cuadrado Myriam,Fernández-Pisonero Isabel,Dosil Mercedes,Sevilla María A.,Montero María J.,Fernández-Salguero Pedro M.,Paramio Jesús M.^ORCID,Bustelo Xosé R.^ORCID

Funder

Fundación Científica Asociación Española Contra el Cáncer (Scientific Foundation, Spanish Association Against Cancer)

Ministerio de Economía y Competitividad (Ministry of Economy and Competitiveness)

Worldwide Cancer Research

Castilla-León Government (CSI049U16). Ramón Areces Foundation (N/A).

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/s41388-018-0433-7.pdf

Reference70 articles.

1. De Craene B, Berx G. Regulatory networks defining EMT during cancer initiation and progression. Nat Rev Cancer. 2013;13:97–110.

2. Dykxhoorn DM, Wu Y, Xie H, Yu F, Lal A, Petrocca F, et al. miR-200 enhances mouse breast cancer cell colonization to form distant metastases. PLoS ONE. 2009;4:e7181.

3. Fischer KR, Durrans A, Lee S, Sheng J, Li F, Wong ST, et al. Epithelial-to-mesenchymal transition is not required for lung metastasis but contributes to chemoresistance. Nature. 2015;527:472–6.

4. Lamouille S, Xu J, Derynck R. Molecular mechanisms of epithelial-mesenchymal transition. Nat Rev Mol Cell Biol. 2014;15:178–96.

5. Nieto MA, Huang RY, Jackson RA, Thiery JP. EMT: 2016. Cell. 2016;166:21–45.

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