The dysfunction of BP180/collagen XVII in keratinocytes promotes melanoma progression-Reference-Cited by-同舟云学术

The dysfunction of BP180/collagen XVII in keratinocytes promotes melanoma progression

Published:2019-08-21 Issue:50 Volume:38 Page:7491-7503
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Hwang Bin-Jin,Zhang Yang,Brozowski Jaime M.,Liu Zhen,Burette Susan,Lough Kendall,Smith Christof C.,Shan Yue,Chen Jinbo,Li Ning,Williams Scott^ORCID,Su Maureen,Googe Paul,Thomas Nancy E.,Liu Zhi

Funder

U.S. Department of Health & Human Services | National Institutes of Health

UNC | University of North Carolina at Chapel Hill

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/s41388-019-0961-9.pdf

Reference58 articles.

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3. Hopkinson SB, Jones JC. The N terminus of the transmembrane protein BP180 interacts with the N-terminal domain of BP230, thereby mediating keratin cytoskeleton anchorage to the cell surface at the site of the hemidesmosome. Mol Biol Cell. 2000;11:277–86.

4. Margadant C, Frijns E, Wilhelmsen K, Sonnenberg A. Regulation of hemidesmosome disassembly by growth factor receptors. Curr Opin Cell Biol. 2008;20:589–96.

5. Leighty L, Li N, Diaz LA, Liu Z. Experimental models for the autoimmune and inflammatory blistering disease, Bullous pemphigoid. Arch Dermatol Res. 2007;299:417–22.

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