VEGF165b, a splice variant of VEGF-A, promotes lung tumor progression and escape from anti-angiogenic therapies through a β1 integrin/VEGFR autocrine loop-Reference-Cited by-同舟云学术

VEGF165b, a splice variant of VEGF-A, promotes lung tumor progression and escape from anti-angiogenic therapies through a β1 integrin/VEGFR autocrine loop

Published:2018-09-07 Issue:7 Volume:38 Page:1050-1066
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Boudria Asma,Abou Faycal Cherine,Jia Tao,Gout Stephanie,Keramidas Michelle,Didier Chloé,Lemaître Nicolas,Manet Sandra,Coll Jean-Luc^ORCID,Toffart Anne-Claire,Moro-Sibilot Denis,Albiges-Rizo Corinne,Josserand Véronique,Faurobert Eva,Brambilla Christian,Brambilla Elisabeth,Gazzeri Sylvie,Eymin Beatrice

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/s41388-018-0486-7.pdf

Reference49 articles.

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3. Roskoski R Jr.. Vascular endothelial growth factor (VEGF) signaling in tumor progression. Crit Rev Oncol Hematol. 2007;62:179–213.

4. Ferrara N, Gerber HP, LeCouter J. The biology of VEGF and its receptors. Nat Med. 2003;9:669–76.

5. Goel HL. Mercurio AM VEGF targets the tumour cell. Nat Rev Cancer. 2013;13:871–82.

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