The BAP1 nuclear deubiquitinase is involved in the nonhomologous end-joining pathway of double-strand DNA repair through interaction with DNA-PK-Reference-Cited by-同舟云学术

The BAP1 nuclear deubiquitinase is involved in the nonhomologous end-joining pathway of double-strand DNA repair through interaction with DNA-PK

Published:2024-02-21 Issue:15 Volume:43 Page:1087-1097
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Sato Hiroki,Ito Tatsuo,Hayashi Takuo,Kitano Shigehisa^ORCID,Erdjument-Bromage Hediye^ORCID,Bott Matthew J.^ORCID,Toyooka Shinichi,Zauderer Marjorie^ORCID,Ladanyi Marc^ORCID

Publisher

Springer Science and Business Media LLC

Link

https://www.nature.com/articles/s41388-024-02966-w.pdf

Reference54 articles.

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2. Bott M, Brevet M, Taylor BS, Shimizu S, Ito T, Wang L, et al. The nuclear deubiquitinase BAP1 is commonly inactivated by somatic mutations and 3p21.1 losses in malignant pleural mesothelioma. Nat Genet. 2011;43:668–72.

3. Hmeljak J, Sanchez-Vega F, Hoadley KA, Shih J, Stewart C, Heiman D, et al. Integrative molecular characterization of malignant pleural mesothelioma. Cancer Discov. 2018;812:1548–65.

4. Harbour JW, Onken MD, Roberson ED, Duan S, Cao L, Worley LA, et al. Frequent mutation of BAP1 in metastasizing uveal melanomas. Science. 2010;330:1410–3.

5. Jensen DE, Proctor M, Marquis ST, Gardner HP, Ha SI, Chodosh LA, et al. BAP1: a novel ubiquitin hydrolase which binds to the BRCA1 RING finger and enhances BRCA1-mediated cell growth suppression. Oncogene. 1998;16:1097–112.

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