XLF and APLF bind Ku80 at two remote sites to ensure DNA repair by non-homologous end joining
Author:
Publisher
Springer Science and Business Media LLC
Subject
Molecular Biology,Structural Biology
Link
http://www.nature.com/articles/s41594-018-0133-6.pdf
Reference68 articles.
1. Lieber, M. R. The mechanism of double-strand DNA break repair by the nonhomologous DNA end-joining pathway. Annu. Rev. Biochem. 79, 181–211 (2010).
2. Chang, H. H. Y., Pannunzio, N. R., Adachi, N. & Lieber, M. R. Non-homologous DNA end joining and alternative pathways to double-strand break repair. Nat. Rev. Mol. Cell Biol. 18, 495–506 (2017).
3. Walker, J. R., Corpina, R. A. & Goldberg, J. Structure of the Ku heterodimer bound to DNA and its implications for double-strand break repair. Nature 412, 607–614 (2001).
4. Grundy, G. J., Moulding, H. A., Caldecott, K. W. & Rulten, S. L. One ring to bring them all—The role of Ku in mammalian non-homologous end joining. DNA Repair (Amst.) 17, 30–38 (2014).
5. Rulten, S. L. & Grundy, G. J. Non-homologous end joining: common interaction sites and exchange of multiple factors in the DNA repair process. Bioessays 39, https://doi.org/10.1002/bies.201600209 (2017).
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