Efficient selenium use by PRDX6 suppresses iron toxicity and ferroptosis
Author:
Publisher
Springer Science and Business Media LLC
Link
https://www.nature.com/articles/s41594-024-01330-6.pdf
Reference6 articles.
1. Stockwell, B. R. Ferroptosis turns 10: Emerging mechanisms, physiological functions, and therapeutic applications. Cell 185, 2401–2421 (2022). A review article that presents mechanisms underlying ferroptosis and its physiological functions.
2. Iwai, K. Regulation of cellular iron metabolism: Iron-dependent degradation of IRP by SCFFBXL5 ubiquitin ligase. Free Radic. Biol. Med. 133, 64–68 (2019). A review article that presents iron homeostasis regulation by FBXL5.
3. Asano, T. et al. Distinct mechanisms of ferritin delivery to lysosomes in iron-depleted and iron-replete cells. Mol. Cell Biol. 31, 2040–2052 (2011). A paper that reports that MEFs are prone to iron toxicity.
4. Driscoll, D. M. & Copeland, P. R. Mechanism and regulation of selenoprotein synthesis. Annu. Rev. Nutr. 23, 17–40 (2003). A review article that presents the mechanisms and regulation of selenoproteins and selenocysteine.
5. Tobe, R. & Mihara, H. Delivery of selenium to selenophosphate synthetase for selenoprotein biosynthesis. Biochim. Biophys. Acta Gen. Subj. 1862, 2433–2440 (2018). A review article that presents possible functions of unidentified selenium carrier protein(s).
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