Structural basis for the context-specific action of the classic peptidyl transferase inhibitor chloramphenicol
Author:
Publisher
Springer Science and Business Media LLC
Subject
Molecular Biology,Structural Biology
Link
https://www.nature.com/articles/s41594-022-00720-y.pdf
Reference52 articles.
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3. Jones, C. N., Miller, C., Tenenbaum, A., Spremulli, L. L. & Saada, A. Antibiotic effects on mitochondrial translation and in patients with mitochondrial translational defects. Mitochondrion 9, 429–437 (2009).
4. Singh, R., Sripada, L. & Singh, R. Side effects of antibiotics during bacterial infection: mitochondria, the main target in host cell. Mitochondrion 16, 50–54 (2014).
5. Li, C. H., Cheng, Y. W., Liao, P. L., Yang, Y. T. & Kang, J. J. Chloramphenicol causes mitochondrial stress, decreases ATP biosynthesis, induces matrix metalloproteinase-13 expression, and solid-tumor cell invasion. Toxicol. Sci. 116, 140–150 (2010).
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