Replication fork stability confers chemoresistance in BRCA-deficient cells-Reference-Cited by-同舟云学术

Replication fork stability confers chemoresistance in BRCA-deficient cells

Published:2016-07-20 Issue:7612 Volume:535 Page:382-387
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Ray Chaudhuri Arnab,Callen Elsa,Ding Xia,Gogola Ewa,Duarte Alexandra A.,Lee Ji-Eun,Wong Nancy,Lafarga Vanessa,Calvo Jennifer A.,Panzarino Nicholas J.,John Sam,Day Amanda,Crespo Anna Vidal,Shen Binghui,Starnes Linda M.,Ruiter Julian R. de,Daniel Jeremy A.,Konstantinopoulos Panagiotis A.,Cortez David,Cantor Sharon B.,Fernandez-Capetillo Oscar,Ge Kai,Jonkers Jos,Rottenberg Sven,Sharan Shyam K.,Nussenzweig André

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/nature18325.pdf

Reference44 articles.

1. Lord, C. J. & Ashworth, A. Mechanisms of resistance to therapies targeting BRCA-mutant cancers. Nature Med. 19, 1381–1388 (2013)

2. Schlacher, K. et al. Double-strand break repair-independent role for BRCA2 in blocking stalled replication fork degradation by MRE11. Cell 145, 529–542 (2011)

3. Schlacher, K., Wu, H. & Jasin, M. A distinct replication fork protection pathway connects Fanconi anemia tumor suppressors to RAD51-BRCA1/2. Cancer Cell 22, 106–116 (2012)

4. Ying, S., Hamdy, F. C. & Helleday, T. Mre11-dependent degradation of stalled DNA replication forks is prevented by BRCA2 and PARP1. Cancer Res. 72, 2814–2821 (2012)

5. Pathania, S. et al. BRCA1 haploinsufficiency for replication stress suppression in primary cells. Nature Commun . 5, 5496 (2014)

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