Prenatal inflammation-induced NF-κB dyshomeostasis contributes to renin-angiotensin system over-activity resulting in prenatally programmed hypertension in offspring-Reference-Cited by-同舟云学术

Prenatal inflammation-induced NF-κB dyshomeostasis contributes to renin-angiotensin system over-activity resulting in prenatally programmed hypertension in offspring

Published:2016-02-15 Issue:1 Volume:6 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Deng Youcai,Deng Yafei,He Xiaoyan,Chu Jianhong,Zhou Jianzhi,Zhang Qi,Guo Wei,Huang Pei,Guan Xiao,Tang Yuan,Wei Yanling,Zhao Shanyu,Zhang Xingxing,Wei Chiming,Namaka Michael,Yi Ping,Yu Jianhua,Li Xiaohui

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep21692.pdf

Reference56 articles.

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2. Laslett, L. J. et al. The worldwide environment of cardiovascular disease: prevalence, diagnosis, therapy and policy issues: a report from the American College of Cardiology. J Am Coll Cardiol 60, S1–49 (2012).

3. Kearney, P. M. et al. Global burden of hypertension: analysis of worldwide data. Lancet 365, 217–223 (2005).

4. Messerli, F. H., Williams, B. & Ritz, E. Essential hypertension. Lancet 370, 591–603 (2007).

5. Barker, D. J. The fetal origins of adult hypertension. J Hypertens Suppl 10, S39–44 (1992).

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