Endothelial cell dysfunction and cytoskeletal changes associated with repression of p16INK4a during immortalization-Reference-Cited by-同舟云学术

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Endothelial cell dysfunction and cytoskeletal changes associated with repression of p16INK4a during immortalization

Published:2012-02-06 Issue:46 Volume:31 Page:4815-4827
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Kan C-Y,Wen V W,Pasquier E,Jankowski K,Chang M,Richards L A,Kavallaris M,MacKenzie K L

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/onc2011645.pdf

Reference63 articles.

1. Bell CD . Endothelial cell tumors. Microsc Res Tech 2003; 60: 165–170.

2. Young RJ, Brown NJ, Reed MW, Hughes D, Woll PJ . Angiosarcoma. Lancet Oncol [Research Support, Non-US Gov't; Review] 2010; 11: 983–991.

3. Tannapfel A, Weihrauch M, Benicke M, Uhlmann D, Hauss J, Wrbitzky R et al. p16INK4A-alterations in primary angiosarcoma of the liver. J Hepatol 2001; 35: 62–67.

4. Weihrauch M, Markwarth A, Lehnert G, Wittekind C, Wrbitzky R, Tannapfel A . Abnormalities of the ARF–p53 pathway in primary angiosarcomas of the liver. Hum Pathol 2002; 33: 884–892.

5. Wagner M, Hampel B, Bernhard D, Hala M, Zwerschke W, Jansen-Durr P . Replicative senescence of human endothelial cells in vitro involves G1 arrest, polyploidization and senescence-associated apoptosis. Exp Gerontol 2001; 36: 1327–1347.

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