Dysregulated TRK signalling is a therapeutic target in CYLD defective tumours-Reference-Cited by-同舟云学术

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Dysregulated TRK signalling is a therapeutic target in CYLD defective tumours

Published:2011-05-09 Issue:41 Volume:30 Page:4243-4260
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Rajan N,Elliott R,Clewes O,Mackay A,Reis-Filho J S,Burn J,Langtry J,Sieber-Blum M,Lord C J,Ashworth A

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/onc2011133.pdf

Reference79 articles.

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2. Alvarez-Rodríguez R, Barzi M, Berenguer J, Pons S . (2007). Bone morphogenetic protein 2 opposes Shh-mediated proliferation in cerebellar granule cells through a TIEG-1-based regulation of Nmyc. J Biol Chem 282: 37170–37180.

3. Annunziata CM, Davis RE, Demchenko Y, Bellamy W, Gabrea A, Zhan F et al. (2007). Frequent engagement of the classical and alternative NF-kappaB pathways by diverse genetic abnormalities in multiple myeloma. Cancer Cell 12: 115–130.

4. April C, Klotzle B, Royce T, Wickham-Garcia E, Boyaniwsky T, Izzo J et al. (2009). Whole-genome gene expression profiling of formalin-fixed, paraffin-embedded tissue samples. PLoS One 4: e8162.

5. Arévalo JC, Waite J, Rajagopal R, Beyna M, Chen Z-Y, Lee FS et al. (2006). Cell survival through Trk neurotrophin receptors is differentially regulated by ubiquitination. Neuron 50: 549–559.

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