JAK2V617F negatively regulates p53 stabilization by enhancing MDM2 via La expression in myeloproliferative neoplasms-Reference-Cited by-同舟云学术

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JAK2V617F negatively regulates p53 stabilization by enhancing MDM2 via La expression in myeloproliferative neoplasms

Published:2011-07-25 Issue:10 Volume:31 Page:1323-1333
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Nakatake M,Monte-Mor B,Debili N,Casadevall N,Ribrag V,Solary E,Vainchenker W,Plo I

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/onc2011313.pdf

Reference49 articles.

1. Akyuz N, Boehden GS, Susse S, Rimek A, Preuss U, Scheidtmann KH et al. (2002). DNA substrate dependence of p53-mediated regulation of double-strand break repair. Mol Cell Biol 22: 6306–6317.

2. Barosi G, Ambrosetti A, Finelli C, Grossi A, Leoni P, Liberato NL et al. (1999). The Italian Consensus Conference on diagnostic criteria for myelofibrosis with myeloid metaplasia. Br J Haematol 104: 730–737.

3. Baxter EJ, Scott LM, Campbell PJ, East C, Fourouclas N, Swanton S et al. (2005). Acquired mutation of the tyrosine kinase JAK2 in human myeloproliferative disorders. Lancet 365: 1054–1061.

4. Berthebaud M, Riviere C, Jarrier P, Foudi A, Zhang Y, Compagno D et al. (2005). RGS16 is a negative regulator of SDF-1-CXCR4 signaling in megakaryocytes. Blood 106: 2962–2968.

5. Bertrand P, Saintigny Y, Lopez BS . (2004). p53's double life: transactivation-independent repression of homologous recombination. Trends Genet 20: 235–243.

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