NAC1 modulates sensitivity of ovarian cancer cells to cisplatin by altering the HMGB1-mediated autophagic response-Reference-Cited by-同舟云学术

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NAC1 modulates sensitivity of ovarian cancer cells to cisplatin by altering the HMGB1-mediated autophagic response

Published:2011-07-11 Issue:8 Volume:31 Page:1055-1064
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Zhang Y,Cheng Y,Ren X,Zhang L,Yap K L,Wu H,Patel R,Liu D,Qin Z-H,Shih I-M,Yang J-M

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/onc2011290.pdf

Reference32 articles.

1. Borst P, Rottenberg S, Jonkers J . (2008). How do real tumors become resistant to cisplatin? Cell Cycle 7: 1353–1359.

2. Cheng Y, Li H, Ren X, Niu T, Hait WN, Yang J . (2010). Cytoprotective effect of the elongation factor-2 kinase-mediated autophagy in breast cancer cells subjected to growth factor inhibition. PLoS One 5: e9715.

3. Davidson B, Berner A, Trope CG, Wang TL, Shih Ie M . (2007). Expression and clinical role of the bric-a-brac tramtrack broad complex/poxvirus and zinc protein NAC-1 in ovarian carcinoma effusions. Hum Pathol 38: 1030–1036.

4. Ellerman JE, Brown CK, de Vera M, Zeh HJ, Billiar T, Rubartelli A et al. (2007). Masquerader: high mobility group box-1 and cancer. Clin Cancer Res 13: 2836–2848.

5. Fanzani A, Zanola A, Rovetta F, Rossi S, Aleo MF . (2011). Cisplatin triggers atrophy of skeletal C2C12 myotubes via impairment of Akt signalling pathway and subsequent increment activity of proteasome and autophagy systems. Toxicol Appl Pharmacol 250: 312–321.

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