IL-21/IL-21R signaling suppresses intestinal inflammation induced by DSS through regulation of Th responses in lamina propria in mice-Reference-Cited by-同舟云学术

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IL-21/IL-21R signaling suppresses intestinal inflammation induced by DSS through regulation of Th responses in lamina propria in mice

Published:2016-08-22 Issue:1 Volume:6 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Wang Yuanyuan,Jiang Xuefeng,Zhu Junfeng,Dan Yue ,Zhang Xiaoqing,Wang Xiao,You Yong,Wang Biao,Xu Ying,Lu Changlong,Sun Xun,Yoshikai Yasunobu

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep31881.pdf

Reference55 articles.

1. Scaldaferri, F. & Fiocchi, C. Inflammatory bowel disease progress and current concepts of etiopathogenesis. J Dig Dis. 8, 171–178 (2007).

2. Xavier, R. J. & Podolsky, D. K. Unravelling the pathogenesis of inflammatory bowel disease. Nature 448, 427–434 (2007).

3. Obermeier, F. et al. Interferon-gamma (IFN-γ)- and tumour necrosis factor (TNF)-induced nitric oxide as toxic effector molecule in chronic dextran sulphate sodium (DSS)-induced colitis in mice. Clin Exp Immunol. 116, 238–245 (1999).

4. Sanchez-Muñoz, F. Role of cytokines in inflammatory bowel disease. World J Gastroenterol. 14, 4280–4288 (2008).

5. Mizoguchi, A., Mizoguchi, E. & Bhan, A. K. The critical role of interleukin 4 but not interferon gamma in the pathogenesis of colitis in T-cell receptor alpha mutant mice. Gastroenterology 116, 320–326 (1999).

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