Interleukin-5 and eosinophils induce airway damage and bronchial hyperreactivity during allergic airway inflammation in BALB/c mice-Reference-Cited by-同舟云学术

Interleukin-5 and eosinophils induce airway damage and bronchial hyperreactivity during allergic airway inflammation in BALB/c mice

Published:1997-06 Issue:3 Volume:75 Page:284-288
ISSN:0818-9641
Container-title:Immunology and Cell Biology
language:en
Short-container-title:Immunol Cell Biol

Author:

Hogan Simon P¹,Koskinen Aulikki¹,Foster Paul S¹

Affiliation:

1. Cellular Signal Transduction Laboratory, Division of Biochemistry and Molecular Biology, The John Curtin School of Medical Research, Australian National University; Canberra Australian Capital Territory Australia

Publisher

Wiley

Subject

Cell Biology,Immunology,Immunology and Allergy

Link

http://www.nature.com/articles/icb199743.pdf

Reference8 articles.

1. Immunological aspects of allergic asthma;Bochner;Annu. Rev. Immunol.,1994

2. Cellular and molecular mechanisms involved in the regulation of eosinophil trafficking in vivo;Hogan;Med. Res. Rev.,1996

3. Interleukin-5 deficiency abolishes eosinophilia. Airways hyperreactivity und lung damage in a mouse asthma model;Foster;J. Exp. Med.,1996

4. InterleLikin-4. but not inlerleukin-5 or eosinophils, is required in a murine model of acute airways hyperreactivity;Corry;J. Exp. Med.,1996

5. Eosinophil recruitment into the respiratory epithelium following antigenic challenge in hyper-IgE mice is accompanied by Interleukin 5-dependent bronchial hyperresponsiveness;Eum;Proc. Natl Acad. Sci. USA,1995

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