Deficient endocannabinoid signaling in the central amygdala contributes to alcohol dependence-related anxiety-like behavior and excessive alcohol intake-Reference-Cited by-同舟云学术

Deficient endocannabinoid signaling in the central amygdala contributes to alcohol dependence-related anxiety-like behavior and excessive alcohol intake

Published:2018-04-05 Issue:9 Volume:43 Page:1840-1850
ISSN:0893-133X
Container-title:Neuropsychopharmacology
language:en
Short-container-title:Neuropsychopharmacol

Author:

Serrano Antonia,Pavon Francisco J.^ORCID,Buczynski Matthew W.,Schlosburg Joel,Natividad Luis A.,Polis Ilham Y.,Stouffer David G.,Zorrilla Eric P.,Roberto Marisa,Cravatt Benjamin F.,Martin-Fardon Rémi^ORCID,Rodriguez de Fonseca Fernando,Parsons Loren H.

Publisher

Springer Science and Business Media LLC

Subject

Psychiatry and Mental health,Pharmacology

Link

http://www.nature.com/articles/s41386-018-0055-3.pdf

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4. Caberlotto L, Rimondini R, Hansson A, Eriksson S, Heilig M. Corticotropin-releasing hormone (CRH) mRNA expression in rat central amygdala in cannabinoid tolerance and withdrawal: evidence for an allostatic shift? Neuropsychopharmacology. 2004;29:15–22.

5. Gilpin NW, Roberto M. Neuropeptide modulation of central amygdala neuroplasticity is a key mediator of alcohol dependence. Neurosci Biobehav Rev. 2012;36:873–88.

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