Neurological dysfunctions in mice expressing different levels of the Q/R site–unedited AMPAR subunit GluR

Neurological dysfunctions in mice expressing different levels of the Q/R site–unedited AMPAR subunit GluR–B

Published:1999-01 Issue:1 Volume:2 Page:57-64
ISSN:1097-6256
Container-title:Nature Neuroscience
language:en
Short-container-title:Nat Neurosci

Author:

Feldmeyer Dirk,Kask Kalev,Brusa Rossella,Kornau Hans–Christian,Kolhekar Rohini,Rozov Andrei,Burnashev Nail,Jensen Vidar,Hvalby Øivind,Sprengel Rolf,Seeburg Peter H.

Publisher

Springer Science and Business Media LLC

Subject

General Neuroscience

Link

http://www.nature.com/articles/nn0199_57.pdf

Reference47 articles.

1. Katz, L. C. & Shatz, C. J. Synaptic activity and the construction of cortical circuits. Science 274, 1133–1138 (1996).

2. Bliss, T. V. & Collingridge, G. L. A synaptic model of memory: long–term potentiation in the hippocampus. Nature 361, 31–39 (1993).

3. Choi, D. W. Calcium: still center–stage in hypoxic–ischemic neuronal death. Trends Neurosci. 18, 58–60 (1995).

4. Rothstein, J. D. & Kuncl, R. W. Neuroprotective strategies in a model of chronic glutamate–mediated motor neuron toxicity. J. Neurochem. 65, 643–651 (1995).

5. Hollmann, M. & Heinemann, S. F. Cloned glutamate receptors. Annu. Rev. Neurosci. 17, 31–108 (1994).

Cited by 212 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. ADAR3 modulates neuronal differentiation and regulates mRNA stability and translation;Nucleic Acids Research;2024-09-01

2. Drugging the Epitranscriptome;Methods and Principles in Medicinal Chemistry;2024-07-05

3. Calcineurin and CK2 Reciprocally Regulate Synaptic AMPA Receptor Phenotypes via α2δ-1 in Spinal Excitatory Neurons;The Journal of Neuroscience;2024-06-17

4. Revisiting Glutamate Excitotoxicity in Amyotrophic Lateral Sclerosis and Age-Related Neurodegeneration;International Journal of Molecular Sciences;2024-05-21

5. Calcineurin regulates synaptic Ca²⁺‐permeable AMPA receptors in hypothalamic presympathetic neurons via α2δ‐1‐mediated GluA1/GluA2 assembly;The Journal of Physiology;2024-04-17