Effects of a genome-wide supported psychosis risk variant on neural activation during a theory-of-mind task-Reference-Cited by-同舟云学术

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Effects of a genome-wide supported psychosis risk variant on neural activation during a theory-of-mind task

Published:2010-03-16 Issue:4 Volume:16 Page:462-470
ISSN:1359-4184
Container-title:Molecular Psychiatry
language:en
Short-container-title:Mol Psychiatry

Author:

Walter H,Schnell K,Erk S,Arnold C,Kirsch P,Esslinger C,Mier D,Schmitgen M M,Rietschel M,Witt S H,Nöthen M M,Cichon S,Meyer-Lindenberg A

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Psychiatry and Mental health,Molecular Biology

Link

http://www.nature.com/articles/mp201018.pdf

Reference70 articles.

1. Allen AJ, Griss ME, Folley BS, Hawkins KA, Pearlson GD . Endophenotypes in schizophrenia: a selective review. Schizophr Res 2009; 109: 24–37.

2. Meyer-Lindenberg A, Weinberger DR . Intermediate phenotypes and genetic mechanisms of psychiatric disorders. Nat Rev Neurosci 2006; 7: 818–827.

3. Mier D, Kirsch P, Meyer-Lindenberg A . Neural substrates of pleiotropic action of genetic variation in COMT: a meta-analysis. Mol Psychiatry 2009; e-pub ahead of print, doi: 10.1038/mp.2009.36.

4. Munafo MR, Brown SM, Hariri AR . Serotonin transporter (5-HTTLPR) genotype and amygdala activation: a meta-analysis. Biol Psychiatry 2008; 63: 852–857.

5. Meyer-Lindenberg A, Zink CF . Imaging genetics for neuropsychiatric disorders. Child Adolesc Psychiatr Clin N Am 2007; 16: 581–597.

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