IL-25 attenuates rheumatoid arthritis through suppression of Th17 immune responses in an IL-13-dependent manner
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/srep36002.pdf
Reference35 articles.
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2. Siebert, S., Tsoukas, A., Robertson, J. & McInnes, I. Cytokines as therapeutic targets in rheumatoid arthritis and other inflammatory diseases. Pharmacological reviews 67, 280–309, doi: 10.1124/pr.114.009639 (2015).
3. Fort, M. M. et al. IL-25 induces IL-4, IL-5, and IL-13 and Th2-associated pathologies in vivo. Immunity 15, 985–995 (2001).
4. Iwakura, Y., Ishigame, H., Saijo, S. & Nakae, S. Functional specialization of interleukin-17 family members. Immunity 34, 149–162, doi: 10.1016/j.immuni.2011.02.012 (2011).
5. Pan, G. et al. Forced expression of murine IL-17E induces growth retardation, jaundice, a Th2-biased response, and multiorgan inflammation in mice. Journal of immunology 167, 6559–6567 (2001).
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