Suppression of CRTC2-mediated hepatic gluconeogenesis by TRAF6 contributes to hypoglycemia in septic shock-Reference-Cited by-同舟云学术

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Suppression of CRTC2-mediated hepatic gluconeogenesis by TRAF6 contributes to hypoglycemia in septic shock

Published:2016-12 Issue:1 Volume:2 Page:
ISSN:2056-5968
Container-title:Cell Discovery
language:en
Short-container-title:Cell Discov

Author:

Lv Sihan,Qiu Xinchen,Li Jian,Li Weida,Zhang Chao,Zhang Zhen-Ning,Luan Bing

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Genetics,Molecular Biology,Biochemistry

Link

http://www.nature.com/articles/celldisc201646.pdf

Reference38 articles.

1. Berk JL, Hagen JF, Beyer WH, Gerber MJ . Hypoglycemia of shock. Ann Surg 1970; 171: 400–408.

2. Ssekitoleko R, Jacob ST, Banura P et al. Hypoglycemia at admission is associated with inhospital mortality in Ugandan patients with severe sepsis. Crit Care Med 2011; 39: 2271–2276.

3. Fischer KF, Lees JA, Newman JH . Hypoglycemia in hospitalized patients. Causes and outcomes. N Engl J Med 1986; 315: 1245–1250.

4. Miller SI, Wallace RJ Jr, Musher DM, Septimus EJ, Kohl S, Baughn RE . Hypoglycemia as a manifestation of sepsis. Am J Med 1980; 68: 649–654.

5. Sugita H, Kaneki M, Tokunaga E et al. Inducible nitric oxide synthase plays a role in LPS-induced hyperglycemia and insulin resistance. Am J Physiol Endocrinol Metab 2002; 282: E386–E394.

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