S-maltoheptaose targets syndecan-bound effectors to reduce smoking-related neutrophilic inflammation
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/srep12945.pdf
Reference40 articles.
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2. Meijer, M., Rijkers, G. T. & van Overveld, F. J. Neutrophils and emerging targets for treatment in chronic obstructive pulmonary disease. Expert Rev Clin Immunol 9, 1055–1068 (2013).
3. Blidberg, K., Palmberg, L., Dahlen, B., Lantz, A. S. & Larsson, K. Increased neutrophil migration in smokers with or without chronic obstructive pulmonary disease. Respirology 17, 854–860 (2012).
4. Kaur, M. & Singh, D. Neutrophil chemotaxis caused by chronic obstructive pulmonary disease alveolar macrophages: the role of CXCL8 and the receptors CXCR1/CXCR2. J Pharmacol Exp Ther 347, 173–180 (2013).
5. Goger, B. et al. Different affinities of glycosaminoglycan oligosaccharides for monomeric and dimeric interleukin-8: a model for chemokine regulation at inflammatory sites. Biochemistry 41, 1640–1646 (2002).
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