Non-classical MHC I-E negatively regulates macrophage activation and Th17 cell development in NOD mice
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/srep12941.pdf
Reference31 articles.
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2. Anderson, M. S. & Bluestone, J. A. The NOD mouse: a model of immune dysregulation. Annual review of immunology 23, 447–485, 10.1146/annurev.immunol.23.021704.115643 (2005).
3. Wicker, L. S., Todd, J. A. & Peterson, L. B. Genetic control of autoimmune diabetes in the NOD mouse. Annual review of immunology 13, 179–200, 10.1146/annurev.iy.13.040195.001143 (1995).
4. Nishimoto, H., Kikutani, H., Yamamura, K. & Kishimoto, T. Prevention of autoimmune insulitis by expression of I-E molecules in NOD mice. Nature 328, 432–434, 10.1038/328432a0 (1987).
5. Lund, T. et al. Prevention of insulin-dependent diabetes mellitus in non-obese diabetic mice by transgenes encoding modified I-A beta-chain or normal I-E alpha-chain. Nature 345, 727–729, 10.1038/345727a0 (1990).
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