A20 (TNFAIP3) deficiency in myeloid cells triggers erosive polyarthritis resembling rheumatoid arthritis-Reference-Cited by-同舟云学术

A20 (TNFAIP3) deficiency in myeloid cells triggers erosive polyarthritis resembling rheumatoid arthritis

Published:2011-08-14 Issue:9 Volume:43 Page:908-912
ISSN:1061-4036
Container-title:Nature Genetics
language:en
Short-container-title:Nat Genet

Author:

Matmati Mourad,Jacques Peggy,Maelfait Jonathan,Verheugen Eveline,Kool Mirjam,Sze Mozes,Geboes Lies,Louagie Els,Guire Conor Mc,Vereecke Lars,Chu Yuanyuan,Boon Louis,Staelens Steven,Matthys Patrick,Lambrecht Bart N,Schmidt-Supprian Marc,Pasparakis Manolis,Elewaut Dirk,Beyaert Rudi,van Loo Geert

Publisher

Springer Science and Business Media LLC

Subject

Genetics

Link

http://www.nature.com/articles/ng.874.pdf

Reference31 articles.

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2. Wertz, I.E. et al. De-ubiquitination and ubiquitin ligase domains of A20 downregulate NF-κB signalling. Nature 430, 694–699 (2004).

3. Boone, D.L. et al. The ubiquitin-modifying enzyme A20 is required for termination of Toll-like receptor responses. Nat. Immunol. 5, 1052–1060 (2004).

4. Hitotsumatsu, O. et al. The ubiquitin-editing enzyme A20 restricts nucleotide-binding oligomerization domain containing 2-triggered signals. Immunity 28, 381–390 (2008).

5. Düwel, M. et al. A20 negatively regulates T cell receptor signaling to NF-κB by cleaving Malt1 ubiquitin chains. J. Immunol. 182, 7718–7728 (2009).

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