MLLT1 YEATS domain mutations in clinically distinctive Favourable Histology Wilms tumours-Reference-Cited by-同舟云学术

MLLT1 YEATS domain mutations in clinically distinctive Favourable Histology Wilms tumours

Published:2015-12 Issue:1 Volume:6 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Perlman Elizabeth J.,Gadd Samantha,Arold Stefan T.,Radhakrishnan Anand,Gerhard Daniela S.,Jennings Lawrence,Huff Vicki,Guidry Auvil Jaime M.,Davidsen Tanja M.,Dome Jeffrey S.,Meerzaman Daoud,Hsu Chih Hao,Nguyen Cu,Anderson James,Ma Yussanne,Mungall Andrew J.^ORCID,Moore Richard A.,Marra Marco A.,Mullighan Charles G.^ORCID,Ma Jing,Wheeler David A.,Hampton Oliver A.,Gastier-Foster Julie M.,Ross Nicole,Smith Malcolm A.

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms10013.pdf

Reference64 articles.

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2. Beckwith, J. B., Kiviat, N. B. & Bonadio, J. F. Nephrogenic rests, nephroblastomatosis, and the pathogenesis of Wilms Tumour. Pediatr. Pathol. 10, 1–36 (1990).

3. Beckwith, J. B. & Palmer, N. F. Histopathology and prognosis of Wilms tumour. Results from the First National Wilms Tumour Study. Cancer 41, 1937–1948 (1978).

4. Bardeesy, N. et al. Anaplastic Wilms tumour, a subtype displaying poor prognosis, harbours p53 gene mutations. Nat. Genet. 7, 91–97 (1994).

5. Gadd, S. et al. Clinically relevant subsets identified by gene expression patterns support a revised ontogenic model of Wilms tumour: a Children's Oncology Group Study. Neoplasia 14, 742–756 (2012).

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