Repositioning tolcapone as a potent inhibitor of transthyretin amyloidogenesis and associated cellular toxicity-Reference-Cited by-同舟云学术

Repositioning tolcapone as a potent inhibitor of transthyretin amyloidogenesis and associated cellular toxicity

Published:2016-02-23 Issue:1 Volume:7 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Sant'Anna Ricardo,Gallego Pablo,Robinson Lei Z.,Pereira-Henriques Alda,Ferreira Nelson,Pinheiro Francisca,Esperante Sebastian,Pallares Irantzu,Huertas Oscar,Rosário Almeida Maria,Reixach Natàlia,Insa Raul,Velazquez-Campoy Adrian,Reverter David,Reig Núria,Ventura Salvador

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms10787.pdf

Reference64 articles.

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3. Westermark, P., Sletten, K., Johansson, B. & Cornwell, G. G. 3rd Fibril in senile systemic amyloidosis is derived from normal transthyretin. Proc. Natl Acad. Sci. USA 87, 2843–2845 (1990).

4. Rapezzi, C. et al. Transthyretin-related amyloidoses and the heart: a clinical overview. Nat. Rev. Cardiol. 7, 398–408 (2010).

5. Hou, X., Aguilar, M. I. & Small, D. H. Transthyretin and familial amyloidotic polyneuropathy. Recent progress in understanding the molecular mechanism of neurodegeneration. FEBS J. 274, 1637–1650 (2007).

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