Diverse drug-resistance mechanisms can emerge from drug-tolerant cancer persister cells-Reference-Cited by-同舟云学术

Diverse drug-resistance mechanisms can emerge from drug-tolerant cancer persister cells

Published:2016-02-19 Issue:1 Volume:7 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Ramirez Michael,Rajaram Satwik,Steininger Robert J.,Osipchuk Daria,Roth Maike A.,Morinishi Leanna S.,Evans Louise,Ji Weiyue,Hsu Chien-Hsiang,Thurley Kevin,Wei Shuguang,Zhou Anwu,Koduru Prasad R.,Posner Bruce A.,Wu Lani F.,Altschuler Steven J.

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms10690.pdf

Reference41 articles.

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4. Bean, J. et al. MET amplification occurs with or without T790M mutations in EGFR mutant lung tumors with acquired resistance to gefitinib or erlotinib. Proc. Natl Acad. Sci. USA 104, 20932–20937 (2007).

5. Bhang, H. E. et al. Studying clonal dynamics in response to cancer therapy using high-complexity barcoding. Nat. Med. 21, 440–448 (2015).

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