Conditional Deletion of Smad1 Ameliorates Glomerular Injury in Progressive Glomerulonephritis

Author:

Araki Makoto,Matsubara Takeshi,Abe Hideharu,Torikoshi Kazuo,Mima Akira,Iehara Noriyuki,Fukatsu Atsushi,Kita Toru,Arai Hidenori,Doi Toshio

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Reference35 articles.

1. Striker, L. J., Doi, T., Elliot, S. & Striker, G. E. The contribution of glomerular mesangial cells to progressive glomerulosclerosis. Semin Nephrol 9, 318–328 (1989).

2. Striker, L. J., Peten, E. P., Elliot, S. J., Doi, T. & Striker, G. E. Mesangial cell turnover: effect of heparin and peptide growth factors. Lab Invest 64, 446–456 (1991).

3. Stokes, M. B., Hudkins, K. L., Zaharia, V., Taneda, S. & Alpers, C. E. Up-regulation of extracellular matrix proteoglycans and collagen type I in human crescentic glomerulonephritis. Kidney Int 59, 532–542 (2001).

4. Abe, H. et al. Type IV collagen is transcriptionally regulated by Smad1 under advanced glycation end product (AGE) stimulation. J Biol Chem 279, 14201–14206 (2004).

5. Matsuda, M. et al. Gene expression of PDGF and PDGF receptor in various forms of glomerulonephritis. Am J Nephrol 17, 25–31 (1997).

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