Bi-allelic inactivation is more prevalent at relapse in multiple myeloma, identifying RB1 as an independent prognostic marker-Reference-Cited by-同舟云学术

Bi-allelic inactivation is more prevalent at relapse in multiple myeloma, identifying RB1 as an independent prognostic marker

Published:2017-02 Issue:2 Volume:7 Page:e535-e535
ISSN:2044-5385
Container-title:Blood Cancer Journal
language:en
Short-container-title:Blood Cancer Journal

Author:

Chavan S S,He J,Tytarenko R,Deshpande S,Patel P,Bailey M,Stein C K,Stephens O,Weinhold N,Petty N,Steward D,Rasche L,Bauer M,Ashby C,Peterson E,Ali S,Ross J,Miller V A,Stephens P,Thanendrarajan S,Schinke C,Zangari M,van Rhee F,Barlogie B,Mughal T I,Davies F E,Morgan G J,Walker B A

Publisher

Springer Science and Business Media LLC

Subject

Oncology,Hematology

Link

http://www.nature.com/articles/bcj201712.pdf

Reference32 articles.

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2. Morgan GJ, Walker BA, Davies FE . The genetic architecture of multiple myeloma. Nat Rev Cancer 2012; 12: 335–348.

3. Weinhold N, Kirn D, Seckinger A, Hielscher T, Granzow M, Bertsch U et al. Concomitant gain of 1q21 and MYC translocation define a poor prognostic subgroup of hyperdiploid multiple myeloma. Haematologica 2016; 101: e116–e119.

4. Walker BA, Boyle EM, Wardell CP, Murison A, Begum DB, Dahir NM et al. Mutational spectrum, copy number changes, and outcome: results of a sequencing study of patients with newly diagnosed myeloma. J Clin Oncol 2015; 33: 3911–3920.

5. Walker BA, Wardell CP, Murison A, Boyle EM, Begum DB, Dahir NM et al. APOBEC family mutational signatures are associated with poor prognosis translocations in multiple myeloma. Nat Commun 2015; 6: 6997.

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