P300-dependent STAT3 acetylation is necessary for angiotensin II-induced pro-fibrotic responses in renal tubular epithelial cells-Reference-Cited by-同舟云学术

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P300-dependent STAT3 acetylation is necessary for angiotensin II-induced pro-fibrotic responses in renal tubular epithelial cells

Published:2014-08-04 Issue:9 Volume:35 Page:1157-1166
ISSN:1671-4083
Container-title:Acta Pharmacologica Sinica
language:en
Short-container-title:Acta Pharmacol Sin

Author:

Ni Jun,Shen Yang,Wang Zhen,Shao De-cui,Liu Jia,Kong Ya-li,Fu Lan-jun,Zhou Li,Xue Hong,Huang Yu,Zhang Wei,Yu Chen,Lu Li-min

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology (medical),Pharmacology,General Medicine

Link

http://www.nature.com/articles/aps201454.pdf

Reference57 articles.

1. Eddy AA . The STAT3 NH2-terminal domain stabilizes enhanceosome assembly by interacting with the p300 bromodomain. Adv Chronic Kidney Dis 2005; 12: 353–65.

2. Liu Y . Renal fibrosis: new insights into the pathogenesis and therapeutics. Kidney Int 2006; 69: 213–7.

3. Matsui F, Rhee A, Hile KL, Zhang H, Meldrum KK . IL-18 induces profibrotic renal tubular cell injury via STAT3 activation. Am J Physiol Renal Physiol 2013; 305: F1014–F1021.

4. Iwano M, Plieth D, Danoff TM, Xue C, Okada H, Neilson EG . Evidence that fibroblasts derive from epithelium during tissue fibrosis. J Clin Invest 2002; 110: 341–50.

5. Liu Y . Epithelial to mesenchymal transition in renal fibrogenesis: pathologic significance, molecular mechanism, and therapeutic intervention. J Am Soc Nephrol 2004; 15: 1–12.

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