METTL3 preferentially enhances non-m6A translation of epigenetic factors and promotes tumourigenesis-Reference-Cited by-同舟云学术

METTL3 preferentially enhances non-m6A translation of epigenetic factors and promotes tumourigenesis

Published:2022-08 Issue:8 Volume:24 Page:1278-1290
ISSN:1465-7392
Container-title:Nature Cell Biology
language:en
Short-container-title:Nat Cell Biol

Author:

Wei Xueju,Huo Yue,Pi Jingnan,Gao Yufeng,Rao Shuan,He Manman,Wei Qinglv,Song Peng,Chen Yiying,Lu Dongxu,Song Wei,Liang Junbo,Xu lingjie,Wang Haixia,Hong Guolin,Guo Yuehong,Si Yanmin,Xu Jiayue,Wang Xiaoshuang,Ma Yanni,Yu Shuyang,Zou Dongling,Jin Jing,Wang Fang,Yu Jia^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology

Link

https://www.nature.com/articles/s41556-022-00968-y.pdf

Reference45 articles.

1. He, P. C. & He, C. m6A RNA methylation: from mechanisms to therapeutic potential. EMBO J. 40, e105977 (2021).

2. Liu, J. et al. A METTL3–METTL14 complex mediates mammalian nuclear RNA N6-adenosine methylation. Nat. Chem. Biol. 10, 93–95 (2014).

3. Fu, Y., Dominissini, D., Rechavi, G. & He, C. Gene expression regulation mediated through reversible m6A RNA methylation. Nat. Rev. Genet. 15, 293–306 (2014).

4. Su, R. et al. METTL16 exerts an mA-independent function to facilitate translation and tumorigenesis. Nat. Cell Biol. 24, 205–216 (2022).

5. Ping, X. L. et al. Mammalian WTAP is a regulatory subunit of the RNA N6-methyladenosine methyltransferase. Cell Res. 24, 177–189 (2014).

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