The dual roles of lymphotoxin-β in promoting breast cancer bone metastasis
Author:
Publisher
Springer Science and Business Media LLC
Link
https://www.nature.com/articles/s41556-024-01479-8.pdf
Reference5 articles.
1. Sosa, M. S., Bragado, P. & Aguirre-Ghiso, J. A. Mechanisms of disseminated cancer cell dormancy: an awakening field. Nat. Rev. Cancer 14, 611–622 (2014). A review article that presents mechanisms of tumour cell dormancy and strategies to target minimal residual disease to prevent metastasis.
2. Satcher, R. L. & Zhang, X. H. Evolving cancer-niche interactions and therapeutic targets during bone metastasis. Nat. Rev. Cancer 22, 85–101 (2022). A review article that presents a summary of the effect of the bone microenvironment on metastasis.
3. Kang, Y. et al. A multigenic program mediating breast cancer metastasis to bone. Cancer Cell 3, 537–549 (2003). This paper reports the identification and validation of genes responsible for osteolytic bone metastasis in breast cancer cells.
4. Wang, H. et al. The osteogenic niche promotes early-stage bone colonization of disseminated breast cancer cells. Cancer Cell 27, 193–210 (2015). This paper reports the interaction between osteoblasts and cancer cells in bone colonization.
5. Lu, X. et al. VCAM-1 promotes osteolytic expansion of indolent bone micrometastasis of breast cancer by engaging alpha4beta1-positive osteoclast progenitors. Cancer Cell 20, 701–714 (2011). This paper reports the role of VCAM1 in metastatic recurrence after dormancy in bone metastasis.
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