Atf3 deficiency promotes genome instability and spontaneous tumorigenesis in mice-Reference-Cited by-同舟云学术

登录注册会员服务联系我们

Atf3 deficiency promotes genome instability and spontaneous tumorigenesis in mice

Published:2017-09-04 Issue:1 Volume:37 Page:18-27
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Wang Z,He Y,Deng W,Lang L,Yang H,Jin B,Kolhe R,Ding H-F,Zhang J,Hai T,Yan C

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/onc2017310.pdf

Reference69 articles.

1. Vousden KH, Prives C . Blinded by the light: the growing complexity of p53. Cell 2009; 137: 413–431.

2. Levine AJ . p53, the cellular gatekeeper for growth and division. Cell 1997; 88: 323–331.

3. Donehower LA, Harvey M, Slagle BL, McArthur MJ, Montgomery CA, Butel JS et al. Mice deficient for p53 are developmentally normal but susceptible to spontanenous tumors. Nature 1992; 356: 215–221.

4. Jacks T, Remington L, Williams BO, Schmitt EM, Halachmi S, Bronson RT et al. Tumor spectrum analysis in p53-mutant mice. Curr Biol 1994; 4: 1–7.

5. Donehower LA, Lozano G . 20 years studying p53 functions in genetically engineered mice. Nat Rev Cancer 2009; 9: 831–841.

Cited by 28 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. The stress-responsive gene ATF3 drives fibroblast activation and collagen production through transcriptionally activating TGF-β receptor Ⅱ in skin wound healing;Archives of Biochemistry and Biophysics;2024-10

2. Pentachloronitrobenzene disturbed murine ventricular wall development by inhibiting cardiomyocyte proliferation via Hec1 downregulation;Science of The Total Environment;2024-02

3. Activating transcription factor 3: A potential therapeutic target for inflammatory pulmonary diseases;Immunity, Inflammation and Disease;2023-09

4. Atf3 defines a population of pulmonary endothelial cells essential for lung regeneration;eLife;2023-05-26

5. Atf3 defines a population of pulmonary endothelial cells essential for lung regeneration;2022-10-14

同舟云学术

1.学者识别学者识别

2.学术分析学术分析

3.人才评估人才评估

"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370

www.globalauthorid.com

TOP

Copyright © 2019-2024 北京同舟云网络信息技术有限公司
京公网安备11010802033243号京ICP备18003416号-3