Entorhinal cortex vulnerability to human APP expression promotes hyperexcitability and tau pathology-Reference-Cited by-同舟云学术

Entorhinal cortex vulnerability to human APP expression promotes hyperexcitability and tau pathology

Published:2024-09-10 Issue:1 Volume:15 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Goettemoeller Annie M.^ORCID,Banks Emmie,Kumar Prateek^ORCID,Olah Viktor J.,McCann Katharine E.,South Kelly,Ramelow Christina C.,Eaton Anna,Duong Duc M.,Seyfried Nicholas T.^ORCID,Weinshenker David^ORCID,Rangaraju Srikant^ORCID,Rowan Matthew J. M.^ORCID

Funder

U.S. Department of Health & Human Services | NIH | National Institute on Aging

Emory University | Alzheimer's Disease Research Center, Emory University

U.S. Department of Health & Human Services | NIH | National Institute of Neurological Disorders and Stroke

Publisher

Springer Science and Business Media LLC

Link

https://www.nature.com/articles/s41467-024-52297-3.pdf

Reference86 articles.

1. Braak, H. & Braak, E. Neuropathological stageing of Alzheimer-related changes. Acta Neuropathol. 82, 239–259 (1991).

2. Gomez-Isla, T. et al. Profound loss of layer II entorhinal cortex neurons occurs in very mild Alzheimer’s disease. J. Neurosci. 16, 4491–4500 (1996).

3. Khan, U. A. et al. Molecular drivers and cortical spread of lateral entorhinal cortex dysfunction in preclinical Alzheimer’s disease. Nat. Neurosci. 17, 304–311 (2014).

4. Kobro-Flatmoen, A., Nagelhus, A. & Witter, M. P. Reelin-immunoreactive neurons in entorhinal cortex layer II selectively express intracellular amyloid in early Alzheimer’s disease. Neurobiol. Dis. 93, 172–183 (2016).

5. Vossel, K. A. et al. Incidence and impact of subclinical epileptiform activity in Alzheimer’s disease. Ann. Neurol. 80, 858–870 (2016).